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Associate Professor, Department of Neurosurgery
Area of Study/Expertise
Molecular and Cellular Neuroscience
Office Location
Robert Wood Johnson Medical School, Daniel I. Kessler Teaching Laboratories, Room N101 675 Hoes Ln W, Piscataway, NJ 08854
Phone Number

Yong Kim, PhD

Robert Wood Johnson Medical School

Associate Professor, Department of Neurosurgery


Dr. Kim earned his PhD degree for the studies of phospholipid lipases-mediated signal transduction at Pohang University of Science and Technology (POSTECH) in Korea. At POSTECH, Dr. Kim received intense biochemical research training from Drs. Sung Ho Ryu and Pann-Ghill Suh, who discovered major isozymes of Phospholipase C (PLC) and pioneered PLC-mediated signal transduction.

Driven by his interest in signal transduction mechanism in nerve cells, Dr. Kim joined the laboratory of Dr. Paul Greengard at the Rockefeller University where he received post-doctoral training in molecular and cellular neuroscience research. After completion of his post-doctoral training, Dr. Kim continued to work in the laboratory as a research group leader and collaborated with Dr. Paul Greengard. While at Rockefeller, Dr. Kim investigated molecular and cellular mechanisms associated with the pathophysiology of major depressive disorder, Alzheimer’s disease, and drug addiction. Dr. Kim has published 51 peer-reviewed articles in the area of neuroscience, biochemistry, and cell biology.

Recently, Dr. Kim started a new laboratory as an Associate Professor in the Department of Neurosurgery, Robert Wood Johnson Medical School at Rutgers University, where he studies the molecular and cellular compensatory or repair mechanisms in the brain by which neurons, neuroglia, and the vasculature system maintain homeostasis in response to brain injury or risk factors of neurodegenerative diseases or psychiatric disorders.


Research Interests:

  • Molecular and cellular mechanisms in the regulation of blood-brain barrier permeability
  • Homeostatic control of neuronal activity or survival mediated by actin regulators
  • Brain cell type-specific compensatory mechanisms in response to traumatic brain injury
  • Molecular and cellular pathways mediating comorbidities in neurological and psychiatric disorders


Publication Highlights:


Laboratory Website: