Research Training Specialist
Dr. Li Ni is a Research Teaching Specialist in the Reynolds Family Spine Laboratory, Department of Neurological Surgery at New Jersey Medical School. Dr. Ni graduated from Beijing Medical University, Beijing, China; and has 5 years of teaching experience in human anatomy and neuroanatomy prior to coming to the USA in the late eighties. Trained as a neuroanatomist, Dr. Ni has extensive experience in vivo and in vitro research techniques, such as immunohistochemistry, molecular biology, small animal surgery, cell culture, etc. Dr. Ni was involved in investigating various aspects of embryonic brain development for the last 15 years. In the laboratory, he performs research on the immune response to acute spinal cord injury in mice.
Related Publications:
A link between plasma membrane calcium ATPase 2 (PMCA2), estrogen and estrogen receptor a signaling in mechanical pain.
Scientific Reports, November 2018
A toll-like receptor 9 antagonist restores below-level glial glutamate transporter expression in the dorsal horn following spinal cord injury.
Scientific Reports, June 2018
Impaired sensitivity to pain stimuli in plasma membrane calcium ATPase 2 (PMCA2)heterozygous mice: a possible modality- and sex-specific role for PMCA2 in nociception.
FASEB Journal, January 2017
Toll like receptor 9 antagonism modulates spinal cord neuronal function and survival: Direct versus astrocyte-mediated mechanisms.
Brain, Behavior and Immunity, August 2016
More publications for Li Ni, M.D.
Related Projects:
- The therapeutic potential of TLR ligands in spinal cord injury
- Proteomic strategies to define new therapeutic targets in animal models of spinal cord injury and disease
- Etiology of multiple sclerosis using an animal model of the disease
- Role of TLR9 in central nervous system development
- Neuron-glia interactions in the spinal cord
- Neuroprotection in spinal cord injury and disease
- Modulation of the inflammatory response in spinal cord injury and disease
- Early surgical decompression for the treatment of acute traumatic spinal cord injury
- The role of plasma membrane calcium ATPase 2 (PMCA2) in the processing of nociceptive and neuropathic pain signals in the spinal cord